ameloblast

Exposure to high levels of fluoride leads to endoplasimic reticulum stress, in which the protein expression of Grp78 has been observed to increase in ameloblast. In addition exposure fluoride causes fluorosis enamel.

Follistatin regulates enamel patterning in mouse incisors by asymmetrically inhibiting BMP signaling and ameloblast differentiation.

There are many variants of ameloblastomas, but the cell type tends to mimic the ameloblast.

Histopathological examination reveals the presence of island and strands of odontogenic epithelium with peripheral columnar cells with palisading nuclei resembling ameloblast and central stellate reticulum-like cells were seen which resembled ameloblastic follicle.

(2009) Ctip2/Bcl11b controls ameloblast formation during mammalian odontogenesis.

INTRODUCTION Ameloblastoma is a tumour of odontogenic epithelial origin and is composed of ameloblast like cells1.

Amelogenin interacts with cytokeratin 5 in ameloblast during enamel growth.

Enamel consists of hydroxyapatite crystallites secreted by ameloblast cells, and crystallites can be nonprismatic, as in most reptiles, or arranged into prisms and interprismatic material, as in most mammals (Koenigswald, 1982; Sahni, 1985).

Monophasic pattern comprises only of clear cell while ameloblastomatous pattern, (6) resembles the growth pattern of ameloblast with nest of cell showing cystic changes and squamous differentiation and peripheral nuclear pallisading with reverse polarity.

(9,10) The undulating course of ameloblast during amelogenesis results in the formation of a specific pattern by so, the course of enamel rods is not the same throughout the thickness of enamel.